Table 4 Activated canonical pathways in NLP-C comparisons
From: Transcriptional landscape of psoriasis identifies the involvement of IL36 and IL36RN
Ingenuity canonical pathways | -log(p-value) | Ratio |
|---|---|---|
Role of IL-17A in Psoriasis | 7.64 | 0.50 |
γ-linolenate Biosynthesis II (Animals) | 4.05 | 0.21 |
Granulocyte Adhesion and Diapedesis | 3.80 | 0.08 |
Role of Hypercytokinemia/hyperchemokinemia in the Pathogenesis of Influenza | 3.62 | 0.15 |
Atherosclerosis Signaling | 3.10 | 0.08 |
Altered T Cell and B Cell Signaling in Rheumatoid Arthritis | 3.09 | 0.09 |
Agranulocyte Adhesion and Diapedesis | 3.02 | 0.07 |
Role of Cytokines in Mediating Communication between Immune Cells | 3.02 | 0.13 |
Differential Regulation of Cytokine Production in Macrophages and T Helper Cells by IL-17A and IL-17F | 2.82 | 0.22 |
LPS/IL-1 Mediated Inhibition of RXR Function | 2.72 | 0.06 |
Stearate Biosynthesis I (Animals) | 2.51 | 0.10 |
Primary Immunodeficiency Signaling | 2.44 | 0.09 |
Differential Regulation of Cytokine Production in Intestinal Epithelial Cells by IL-17A and IL-17F | 2.41 | 0.17 |
IL-17 Signaling | 2.34 | 0.09 |
Communication between Innate and Adaptive Immune Cells | 2.33 | 0.07 |
Uracil Degradation II (Reductive) | 2.32 | 0.18 |