Fig. 1From: Fine mapping of QTL and genomic prediction using allele-specific expression SNPs demonstrates that the complex trait of genetic resistance to Marek’s disease is predominantly determined by transcriptional regulationAmount of genetic variation accounted for by the polygenic effect vs. each SNP panel. The data was fit using a mixed model containing both a polygenic effect, associated with the pedigree relationship matrix, and a SNP panel, associated with either 1824 ASE SNPs or 3097 random SNPsBack to article page