Skip to main content

Table 2 Gene ontology (GO) enrichment of consensus ROH analysisa

From: Genomic analysis of Ugandan and Rwandan chicken ecotypes using a 600 k genotyping array

GO: ID Go: term
GO:0006915 Apoptotic process
GO:0002209 Behavioral defense response
GO:0001662 Behavioral fear response
GO:0070588 Calcium ion transmembrane transport
GO:0071345 Cellular response to cytokine stimulus
GO:0071495 Cellular response to endogenous stimulus
GO:0071396 Cellular response to lipid
GO:0033554 Cellular response to stress
GO:0019221 Cytokine-mediated signaling pathway
GO:0006281 DNA repair
GO:0007631 Feeding behavior
GO:0007599 Hemostasis
GO:0031663 Lipopolysaccharide-mediated signaling pathway
GO:0032873 Negative regulation of stress-activated MAPK cascade
GO:0070303 Negative regulation of stress-activated protein kinase signaling cascade
GO:0016310 Phosphorylation
GO:0042981 Regulation of apoptotic process
GO:0080135 Regulation of cellular response to stress
GO:0001959 Regulation of cytokine-mediated signaling pathway
GO:0043408 Regulation of MAPK cascade
GO:2000377 Regulation of reactive oxygen species metabolic process
GO:0080134 Regulation of response to stress
GO:0032319 Regulation of rho gtpase activity
GO:1901700 Response to oxygen-containing compound
GO:0009314 Response to radiation
GO:0006950 Response to stress
GO:0023014 Signal transduction by phosphorylation
GO:0033209 Tumor necrosis factor-mediated signaling pathway
GO:0042060 Wound healing
GO:0003684 Damaged DNA binding
GO:0005246 Calcium channel regulator activity
GO:0005488 Binding
GO:0008289 Lipid binding
GO:0030234 Enzyme regulator activity
GO:0005509 Calcium ion binding
GO:1901363 Heterocyclic compound binding
GO:0005160 Transforming growth factor beta receptor binding
GO:0005543 Phospholipid binding
GO:0004674 Protein serine/threonine kinase activity
GO:0035258 Steroid hormone receptor binding
GO:0008083 Growth factor activity
  1. Subset of GO terms that could putatively affect responses to environmental stressors. aAll terms were statistically significant at FDR < 0.15. This analysis lends evidence to support that all three populations experienced selective pressures for variants effecting stress response, immune response, and behavior