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Fig. 8 | BMC Genomics

Fig. 8

From: Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

Fig. 8

Gene interaction networks controlling the proteasome (a) and cell signaling via G-protein coupled receptors (b) in liver of riboflavin-deficient and riboflavin-rescued embryos at e15. Panel a shows interactions between components of the proteasome and direct target genes of cyclin D (CCND1) and annotated by IPA as “Neurological Disease”. The gene network in Panel b depicts interactions of several G-protein coupled receptors (GPCRs) and members of the tublin complex. This network was functionally annotated by IPA as related to “Hematological Disease” and “Cell Signaling”

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