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Table 1 Summary statistics of TAD mapping

From: Putative bovine topological association domains and CTCF binding motifs can reduce the search space for causative regulatory variants of complex traits

Input TADs Reference assembly Number of TADs Mean TAD width (kb) Number of input TADs mapped (ratio)
Study Cell or tissue 1 location in bovine genome Same bovine chromosome Bovine genome
Dixon 2012 hESC hg18 3127 852.2
bostau6 2885 830.5 2784(89.03%) 2930(93.70%) 2956(94.53%)
IMR90 hg18 2349 1122
bostau6 2236 1071 2050(87.27%) 2198(93.57%) 2261(96.25%)
mESC mm9 2200 1093
bostau6 2173 1104 1912(86.91%) 2041(92.77%) 2127(96.68%)
cortex mm9 1519 1542
bostau6 1597 1489 1283(84.46%) 1401(92.23%) 1502(98.88%)
Rudan 2015 liver mm10 3643 695
bostau6 3507 602.5 2388(65.55%) 2873(78.86%) 2901(79.63%)
canfam3 3315 686.5
bostau6 2979 810 2731(82.38%) 2887(87.09%) 2916(87.96%)
  1. The source study and cell/tissue from input TAD dataset, and also the input and output reference assemblies with the number of TADs and their mean width (in kilobases) are shown. Also presented are the number of input TADs that did not split during mapping (1 location in the bovine genome), the number of input TADs that did not split or split intra-chromosomally during mapping (same bovine chromosome), and the number of input TADs that did not split, split intra-chromosomally or inter-chromosomally during mapping (bovine genome)