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Table 4 Loss-of-function mutation in E. tarda strains E22 and NUF806

From: Comparative genomics reveals that a fish pathogenic bacterium Edwardsiella tarda has acquired the locus of enterocyte effacement (LEE) through horizontal gene transfer

Gene product Gene Length (bp)* Nonsynonymous mutation INDEL Type
Strain E22      
  hypothetical protein ETAE_0831 1668 TGG(W)998TAG(*)   Nonsense
  two-component response regulator esrB 645 TCG(S)374TAG(*)   Nonsense
  putative NADH:flavin oxidoreductase/NADH oxidase ETAE_0969 2067 TGT(C)1116TGA(*)   Nonsense
  ferric enterobactin transport protein fepE 1047 AAC(N)134AGC(S) GCGGC992 Frameshift
  phospholipase D family protein ETAE_1290 1245   G880 Frameshift
  putative exoprotein-precursor ETAE_2088 825   G809 Frameshift
  bifunctional glutathionylspermidine ETAE_2689 348 GCG(A)331TCG(S) 332CGCCGGT Frameshift
  cobyrinic acid ac-diamide synthase ETAE_2747 132   C119 Frameshift
  bifunctional chorismate mutase/prephenate dehydrogenase tyrA 1122   C524 Frameshift
Strain NUF806      
  hypothetical protein ETAE_0339 120   78T Frameshift
  dihydropyrimidinase ygeZ 1386   962G Frameshift
  1. * Lengths of EIB202 genes.