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Table 4 Loss-of-function mutation in E. tarda strains E22 and NUF806

From: Comparative genomics reveals that a fish pathogenic bacterium Edwardsiella tarda has acquired the locus of enterocyte effacement (LEE) through horizontal gene transfer

Gene product

Gene

Length (bp)*

Nonsynonymous mutation

INDEL

Type

Strain E22

     

  hypothetical protein

ETAE_0831

1668

TGG(W)998TAG(*)

 

Nonsense

  two-component response regulator

esrB

645

TCG(S)374TAG(*)

 

Nonsense

  putative NADH:flavin oxidoreductase/NADH oxidase

ETAE_0969

2067

TGT(C)1116TGA(*)

 

Nonsense

  ferric enterobactin transport protein

fepE

1047

AAC(N)134AGC(S)

GCGGC992

Frameshift

  phospholipase D family protein

ETAE_1290

1245

 

G880

Frameshift

  putative exoprotein-precursor

ETAE_2088

825

 

G809

Frameshift

  bifunctional glutathionylspermidine

ETAE_2689

348

GCG(A)331TCG(S)

332CGCCGGT

Frameshift

  cobyrinic acid ac-diamide synthase

ETAE_2747

132

 

C119

Frameshift

  bifunctional chorismate mutase/prephenate dehydrogenase

tyrA

1122

 

C524

Frameshift

Strain NUF806

     

  hypothetical protein

ETAE_0339

120

 

78T

Frameshift

  dihydropyrimidinase

ygeZ

1386

 

962G

Frameshift

  1. * Lengths of EIB202 genes.