Figure 7

A putative action model for MES-treatment inducing male sterility. Some important functions and genes affected by MES treatment in leaf tissue (left rectangle) and developing anther tissues (right rectangle) are listed (see text for details). Two vertical dashed lines in the right rectangle separate three anther tissues. ‘↑’ after function categories or genes means up-regulation; ‘↓’ after function categories or genes means down-regulation. Two aspects of putative reasons for carbohydrate and lipid metabolism alteration were showed by dashed arrows ‘?’ represents unclear MES transport pathway. MES, Monosulfuron Ester Sodium; ALS, acetolactate synthase; BCAAs: Branch-Chain Amino Acids; AGP, ADP glucose pyrophosphorylase; PMG, Phosphoglucomutase; DPE1, Disproportionating enzyme; PHS2, alpha-glucan phosphorylase 2; SWEET 11, Nodulin MtN3 family protein; BXL1, beta-xylosidase; pectin lyases, pectin lyase superfamily protein; VGDH2, VANGUARD 1 homolog 2; PPME1: Pectin lyase-like superfamily protein; FLA5: FASCICLIN-like arabinogalactan protein 5 precursor; VGDH1: Plant invertase/pectin methylesterase inhibitor; VGD1: Plant invertase/pectin methylesterase inhibitor; PGA4, Polygalacturonase 4; UGE3, UDP-D-glucose/UDP-D-galactose 4-epimerase 3; HA9, H(+)-ATPase 9.