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BMC Genomics

Open Access

Erratum to: Genome sequencing of the Trichoderma reesei QM9136 mutant identifies a truncation of the transcriptional regulator XYR1 as the cause for its cellulase-negative phenotype

  • Alexander Lichius1,
  • Frédérique Bidard2,
  • Franziska Buchholz1,
  • Stéphane Le Crom3,
  • Joel Martin4,
  • Wendy Schackwitz4,
  • Tina Austerlitz1,
  • Igor V. Grigoriev4,
  • Scott E. Baker5,
  • Antoine Margeot2,
  • Bernhard Seiboth1Email author and
  • Christian P. Kubicek1
Contributed equally
BMC Genomics201516:725

https://doi.org/10.1186/s12864-015-1917-2

Received: 10 September 2015

Accepted: 10 September 2015

Published: 22 September 2015

The original article was published in BMC Genomics 2015 16:326

Erratum to: BMC Genomics doi 10.1186/s12864-015-1526-0

Following the publication of our recent article in BMC Genomics [1] we wish to bring the following corrigendum to your attention. In the above paper, we wrote in the discussion on page 13: “These are V756F in XlnR (corresponding to V801 in XYR1) and A804V (based on our analysis; not A824V as stated by the authors) in XYR1”. Our analysis of the XYR1 sequence was based on the available Trichoderma reesei QM6a XYR1 sequences deposited in the NCBI database [Protein Accession Number XP_006966092.1 and EGR48040.1]. Recently, Derntl et al. [2] identified that the second intron in xyr1 is in fact not spliced thus giving rise to a protein that, while maintaining the reading frame, is 20 amino acids longer. Consequently, the position A824V given by Derntl et al. [2] is correct, and the numbering of amino acids in our paper after G319 has to be increased by 20.

A corrected version of our statement would read: "These are V756F in XlnR of A. niger which corresponds to V821 in T. reesei XYR1, as well as A824V in XYR1 of T. reesei [35, 42]".

We apologize for this mistake, but would like to stress that none of the results or conclusions in our paper are affected by this change.

Notes

Declarations

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors’ Affiliations

(1)
Research Division Biotechnology and Microbiology, Institute of Chemical Engineering, Vienna University of Technology
(2)
IFP Energies Nouvelles
(3)
Sorbonne Universités, UPMC Université Paris 06, Institut de Biologie Paris Seine (IBPS), FR 3631, Département des Plateforme
(4)
US Department of Energy Joint Genome Institute
(5)
Environmental Molecular Sciences Laboratory, Pacific Northwest National Laboratory

References

  1. Lichius A, Bidard F, Buchholz F, Le Crom S, Martin J, Schackwitz W, et al. Genome sequencing of the Trichoderma reesei QM9136 mutant identifies a truncation of the transcriptional regulator XYR1 as the cause for its cellulase-negative phenotype. BMC Genomics. 2015;16:326.PubMed CentralView ArticlePubMedGoogle Scholar
  2. Derntl C, Gudynaite-Savitch L, Calixte S, White T, Mach RL, Mach-Aigner AR. Mutation of the xylanase regulator 1 causes a glucose blind hydrolase expressing phenotype in industrially used Trichoderma strains. Biotechnol Biofuels. 2013;6:62.PubMed CentralView ArticlePubMedGoogle Scholar

Copyright

© Lichius et al. 2015

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